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The increased risk of atherosclerosis in diabetes mellitus cannot be totally reversed by the treatment of classical risk factors such as for example high cholesterol, hypertension, obesity and smoking. Recent reports on the higher predictive value of blood glucose and triglycerides after meals suggest that important proatherogenic processes can occur during the postprandial state. In this project we will focus on the interactions between postprandial lipids, monocytes and vitamin E. The ultimate aim is to set up clear guidelines on optimal intakes of lipids and antioxidants and thus to restrict cardiovascular risk.
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