plasmamembran – English Translation – Keybot Dictionary

Spacer TTN Translation Network TTN TTN Login Deutsch Français Spacer Help
Source Languages Target Languages
Dictionary
&nbsp;<a href='https://iate.europa.eu/home'>IATE</a> 2841
Plasmamembran => plasmalemma
&nbsp;<a href='https://iate.europa.eu/home'>IATE</a> 00
Plasmamembran => plasma membrane

Keybot      67 Results   18 Domains
  www.hochschildmining.com  
Show textShow cached sourceOpen source URL
Plasmamembran
Compare text pagesCompare HTM pagesOpen source URLOpen target URLDefine bildwoerterbuch.com as primary domain
plasma membrane
  2 Hits www.tierorthopaedie-frankfurt.de  
Show textShow cached sourceOpen source URL
plasmamembran
Compare text pagesCompare HTM pagesOpen source URLOpen target URLDefine junior.edumedia-sciences.com as primary domain
plasma membrane
  www.aanemane.org  
Show textShow cached sourceOpen source URL
für Pflanzenzellen. Besonders spannend sind Peptide, weil man diese für eine ganz bestimmte Zielstruktur zuschneiden kann. Doch wie bekommt man Peptide durch Zellwand und Plasmamembran in die Zelle hinein?
Compare text pagesCompare HTM pagesOpen source URLOpen target URLDefine botanik.kit.edu as primary domain
for plant cells. Especially interesting are peptides, because they can be tailored for specific cellular Targets. But how to get those peptides through cell wall and cell membrane into the cell?
  3 Hits ist.ac.at  
Show textShow cached sourceOpen source URL
Kalziumionen strömen schnell in das präsynaptische Terminal ein und die Kalziumkonzentration im präsynaptischen Terminal steigt. Das erlaubt synaptischen Vesikel, gefüllt mit Neurotransmitter, mit der Plasmamembran zu verschmelzen und den Neurotransmitter in den synaptischen Spalt freizusetzen.
Compare text pagesCompare HTM pagesOpen source URLOpen target URLDefine ist.ac.at as primary domain
At a chemical synapse, signal transmission requires an elaborate sequence of events. It starts when an electrical signal, the action potential, reaches the synaptic terminal of the presynaptic neuron. This causes voltage-gated calcium channel to open. Calcium ions rapidly stream into the presynaptic terminal and the calcium concentration in the presynaptic terminal rises. This allows synaptic vesicles filled with neurotransmitter to fuse with the plasma membrane and release the neurotransmitters into the synaptic cleft. Speed is essential in information transmission. Therefore, before the action potential even arrives at the presynaptic terminal, vesicles containing neurotransmitter line up in a fusion-ready state at docking sites in the presynaptic terminal. When the action potential reaches the presynaptic terminal, the vesicles can rapidly fuse and release the neurotransmitter. Functionally, docking sites limit the maximum number of vesicles that can be released at each action potential, this determines the strength of the synapse. Until now, a clear link between the functional aspect of docking sites and their morphological aspect as sites where vesicles dock could not be established in the mammalian brain.
  www.tsc-silos.com  
Show textShow cached sourceOpen source URL
Neben den klassischen Protein-Rezeptoren spielen Protein- und Lipid-gebundene Glykostrukturen eine wichtige Rolle bei der Zellerkennung. Die großen Oligosaccharidbestandteile der Glykokonjugate bilden dabei eine Zuckerschicht auf der extrazellulären Seite der Plasmamembran, die als Glykocalyx bezeichnet wird.
Compare text pagesCompare HTM pagesOpen source URLOpen target URLDefine ioc.kit.edu as primary domain
Individual surface properties of cells are essential for the recognition of many vital factors. These factors include whole cells, proteins, growth factors and messenger substances as well as pathogens and drugs. The recognition of such factors is highly specific and individual for every cell type. It is often based on interactions with corresponding receptors on the extracellular site of the cell membrane. Besides the classical protein receptors protein- and lipid-bound glycostructures play an important role in cell recognition. Large oligosaccharide components of glycoconjugates form a sugar layer on the extracellular site of the plasma membrane, the so-called glycocalyx. These sugar structures can be labeled in the living cell by copper-free click-reactions. Therefore the cells are incubated with synthetic sugars that include azido groups. The synthetic sugar molecules are incorporated in the glycostructures of the cell and cyclooctynes that carry fluorophores can be bound to the azido groups selectively by a copper-free click-reaction. The development of sugar structures, e.g. during the embryonic development, can be monitored by reactions on a staggered basis of azido groups with cyclooctynes carrying different fluorophores.
  2 Hits www.dkfz.de  
Show textShow cached sourceOpen source URL
Nekrose wird zwar als passive Form des Zelltods verstanden, teilt jedoch viele der Signaltransduktionswege mit dem programmierten Zelltod. Im Gegensatz zur Apoptose löst die Nekrose aufgrund der Zerstörung der Plasmamembran und der Freisetzung von cytosolischen Komponenten Entzündungen aus.
Compare text pagesCompare HTM pagesOpen source URLOpen target URLDefine dkfz.de as primary domain
We are investigating the control and crosstalk between programmed cell death (PCD) mechanisms of apoptosis, autophagy and necrosis in pancreatic, breast and brain cancer cells. Apoptosis, the most studied PCD mode (Type I), is activated by either the death receptor or the mitochondrial pathway. Autophagy is a process by which intracellular components are sequestered by autophagosomes, which then fuse with and are degraded by lysosomes. In the cancer cell autophagy can paradoxically act as either an alternative cell death pathway (Type II PCD) or as a potent survival response to stress, e.g. hypoxia and chemotherapies. Although considered a passive cell death, many pathways are common to necrosis and PCD modes. In contrast to apoptosis, necrosis is inflammatory due to the rupture of the plasma membrane and the release of specific cytosolic components. As apoptosis does not generate an immune response, the strict focus on apoptosis-inducing therapies may not be fully productive. Our research is aimed at revealing how individual pathway activities and crosstalk between PCD pathways can be tuned to optimize intrinsic and extrinsic pancreatic cancer cell death.
  4 Hits dukelanguage.com  
Show textShow cached sourceOpen source URL
Ist die Impulsanstiegszeit größer als die Aufladezeit der Plasmamembran, wird die elektrische Feldstärke vollständig in die Membran verdrängt. Das Zellinnere bleibt zunächst feldfrei. Die Membran, die aus elektrischer Sicht einen Isolator darstellt, wird dabei aufgeladen.
Compare text pagesCompare HTM pagesOpen source URLOpen target URLDefine ihm.kit.edu as primary domain
For slowly rising and low-amplitude field pulses, only the plasma-membrane of the cells, which can be considered as an electrical isolator, is affected. Initially, the electric field in the cell interior is zero. Due to the external electric field, the cell´s plasma-membrane is charged. Elevated transmembrane voltages cause a structural reorganization of membrane constituents, i.e. the phospolipid molecules, which results in an increase of membrane permeability by formation of aqueous pores, see illustration to the right. This process is called electroporation. It is applied for exchanging water-soluble substances across the cell boundary.